A dictionary of neurological signs : clinical neurosemiology by A.J. Larner
By A.J. Larner
While such a lot textbooks of neurology strategy the topic from both a symptomatic or a diagnostic point of view, this dictionary specializes in many of the neurological symptoms that could be elicited via history-taking and actual exam. greater than six hundred dependent and cross-referenced definitions are supplemented, the place acceptable, with the neuroanatomical and neurophysiological foundation of every signal. as a result the semiological price of indicators is elucidated in a manner which makes an attempt to combine scientific phenomenology with underlying neurobiology, therefore guiding anatomical and pathological prognosis and acceptable therapy. This e-book should be of curiosity to all scholars of neurology, undergraduate and postgraduate, in addition to more matured clinicians, together with basic physicians and common practitioners, and individuals of ancillary professions who take care of sufferers with neurological disease. Read more...
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Extra resources for A dictionary of neurological signs : clinical neurosemiology
Alloaesthesia Allodynia posterior cerebral artery occlusion (sensory variant) following commissurotomy (corpus callosotomy alone insufficient). Functional imaging studies in corticobasal degeneration, along with the evidence from focal vascular lesions, suggest that damage to and/or hypometabolism of the medial frontal cortex (Brodmann area 32) and the supplementary motor area (Brodmann area 6) are associated with alien limb phenomena. More generally, it seems that these areas are involved in the execution of learned motor programs, and damage thereto may lead to the release of learned motor programs from voluntary control.
The anatomical substrates of attention encompass the ascending reticular activating system of the brainstem, the thalamus, and the prefrontal (multimodal association) cerebral cortex (especially on the right). Damage to any of these areas may cause impaired attention. Attention may be tested in a variety of ways, all of which are essentially looking for a defect in working memory (“short term memory”): Orientation in time/place; Digit span forwards/backwards; Reciting months of the year backwards, counting back from 30 to 1; Serial sevens (serial subtraction of 7 from 100, = 93, 86, 79, 72, 65).
Alzheimer’s disease) but sometimes in isolation (primary progressive aphasia). Albert ML. Treatment of aphasia. Archives of Neurology 1998; 55: 1417-9 Benson DF, Ardila A. Aphasia: a clinical perspective. New York: OUP 1996 Damasio AR. Aphasia. New England Journal of Medicine 1992; 326: 531-9 Mohr JP. Acquired language disorders. ). Diseases of the Nervous System: Clinical Neurobiology. London: Heinemann 1986:816-27 Willmes K, Poeck K. To what extent can aphasic syndromes be localized? Brain 1993; 116: 1527-40 [Cross References: AGRAMMATISM; AGRAPHIA; ALEXIA; ANOMIA; APROSODIA, APROSODY; BROCA’S APHASIA; CIRCUMLOCUTION; CONDUCTION APHASIA; CONDUIT D’APPROCHE; DYSARTHRIA; DYSPROSODY; JARGON APHASIA; NEOLOGISM; OPTIC APHASIA; PARAPHASIA; TRANSCORTICAL APHASIA; WERNICKE’S APHASIA] Aphemia Aphemia is a motor disorder of speech production with preserved comprehension of spoken and written language.