Mean Genes: From Sex to Money to Food Taming Our Primal by Terry Burnham, Jay Phelan
By Terry Burnham, Jay Phelan
Why can we want-and do-so many stuff which are undesirable for us? In Mean Genes Terry Burnham and Jay Phelan argue that we have to cease trying to Sigmund Freud for solutions and begin seeking to Charles Darwin. Mean Genes unearths that our struggles for self-improvement are, in reality, battles opposed to our personal genes-genes that helped our far-off ancestors flourish, yet are egocentric and misplaced within the sleek global. utilizing this evolutionary lens, Mean Genes brilliantly examines the problems that the majority have an effect on our lives-body photograph, cash, dependancy, violence, and relationships, friendship, love, and fidelity-and deals steps to aid us lead extra pleasurable lives.
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Additional info for Mean Genes: From Sex to Money to Food Taming Our Primal Instincts
Goldstein: It doesn’t hurt you to do so, it just doesn’t help you. Abbas: If it is a collection of common variants that di¡er from one person to another, then looking at these homogeneous populations is not going to be informative. Daly: The advantages they might have are factors such as common environment and diet, not of common genetic history. Kere: But you are not worse o¡. 44 DISCUSSION Abbas: If you have to choose a patient population, you have to start with the assumption that it is a collection of common variants, but would you choose a more heterogeneous population?
In cases where sperm typing has been done, for example, these methods appear to be quite accurate. Vyse: I wanted to follow up on the question about susceptibility to infection. The genetics of susceptibility to infection and the genetics of the susceptibility to autoimmune disease are strongly related. These are common issues. We have some data that re£ects this. The in£uence of Fcg receptor polymorphism is related, for example, to lupus. There is evidence to suggest that this may in£uence the resistance or otherwise of mice within wild-type populations to infection.
But what is the cause of common autoimmune diseases? We need to get to this question: are there collections of common variants? Rioux: It is biology. It is a spectrum; it is not one extreme or the other. Goodnow: Is the common variant hypothesis an unfalsi¢able hypothesis in a Popperian sense? Wakeland: Many autoimmune disease alleles are very common. The best example at the moment would be disease-associated HLA alleles. All these alleles are relatively common, globally distributed and are very powerful as disease predisposing elements.