Mechanisms in Myeloid Tumorigenesis 1988: Workshop at the by Archibald S. Perkins (auth.), Grace L. C. Shen-Ong Ph. D.,
By Archibald S. Perkins (auth.), Grace L. C. Shen-Ong Ph. D., Michael Potter M. D., Neal G. Copeland Ph. D. (eds.)
Contents: category of Myeloid Tumors.- particular Chromosomal Abnormalities in Human and Murine Myeloid Hematologic Malignancies.- Disruption of "Oncogenes" via Virus Insertion in Murine Myeloid Tumorigenesis.- Myeloid Leukemogenesis precipitated via Acute reworking Virus Constructs in In Vitro and In Vivo version Systems.- particular Genes fascinated about Myeloid Tumorigenesis.- Proposed Mechanisms in Myeloid Tumorigenesis.
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Extra resources for Mechanisms in Myeloid Tumorigenesis 1988: Workshop at the National Cancer Institute, National Institutes of Health, Bethesda, MD, USA, March 22, 1988
Polycythemia Vera PV is the second most extensively studied MPD. Of untreated and treated PV patients (excluding patients analyzed only in the leukemic blast phase), one-fourth (23%) have chromosomally abnormal clones in their initial analysis. Abnormalities are less common in untreated patients than in those who have been treated with cytotoxic agents prior to their first cytogenetic examination (14% versus 39%, respectively). The incidence of abnormal clones is even higher (85%) in treated PV patients whose disease has transformed to a leukemic phase.
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In: King RA, Rotter JJ, Motulsky AG (eds) The Genetic Basis of Common Diseases. Oxford University Press, New York, in press Mitelman F (1988) Catalog of Chromosome Aberrations in Cancer (3rd edition). Alan R. Liss, New York, p 1 Rowley JD (1984) Biological implications of consistent chromosome rearrangements in leukemia and lymphoma. Cancer Res 44:3159-3165 Second MIC Cooperative study Group (1988) Morphologic, immunologic and cytogenetic (MIC) working classification of the acute myeloid 1eukaemias.