The Mysterious Origins of Hybrid Man: Crossbreeding and the by Susan B. Martinez

By Susan B. Martinez

"Martinez's leading edge and scalpel-like study upends the academy's loved (but sincerely warped) views on evolution and humanoid history."—(Paul Von Ward, writer of "We've by no means Been Alone" and "The Soul Genome") "Susan B. Martinez, Ph.D., has written a unprecedented publication. She has given all those that think about the origins of humankind a unprecedented reward of remarkably thorough research."—(Brad Steiger, writer "Worlds ahead of Our Own" and "Conspiracies and mystery Societies: the whole Dossier") Did we evolve from apes, or are all of us descendants of Adam and Eve? Why is the "missing link" nonetheless lacking? Is the dumb good fortune of normal choice legitimate? Piecing jointly the protohistory of humanity via anthropology, genetics, paleolinguistics, and indigenous traditions, Susan B. Martinez deals a wholly unique substitute to Darwin's evolution: glossy humanity didn't evolve yet is a mosaic of combined ancestry, the results of eons of cross-breeding and retro-breeding between various teams, together with Cro-Magnon, Neanderthal, hobbits, giants, and Africa's "Lucy" and "Zinj." Martinez exhibits that there have been a number of "Gardens of Eden" and the way every one continent had its personal mixture of races ahead of the good Flood, which prompted the diaspora that introduced a renaissance of tradition to each department of the Earth. Martinez explains Homo sapiens' psychological powers (the nice breakthrough) in cosmological terms—how we're the made from either heaven and earth. She identifies the "Sons of Heaven" and the angel-engendered races, explaining how Homo sapiens got the "blood of the gods," which endowed us with a soul. delivering the last word answer to the Evolution as opposed to Creationism debate, this landmark examine of hybrid guy justifies his suddenly surprising visual appeal within the fossil list, the curious parallels among oral histories of the world's humans, and why anatomically sleek beneficial properties are present in the earliest paleontological proof. SUSAN B. MARTINEZ, Ph.D., earned her doctorate in anthropology at Columbia collage, the place she additionally served as a lecturer in ethnolinguistics. She is the publication overview editor on the Academy of religious and attention stories and the writer of "The Psychic lifetime of Abraham Lincoln", "The Hidden Prophet", "Time of the Quickening", and "The misplaced background of the Little People". A contributor to "Forbidden Science" and "Darklore", her paintings additionally seems in "Atlantis Rising", "FATE", and "New Dawn" magazines. She lives in Clayton, Georgia.

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PMP22 DISORDERS WITH TRINUCLEOTIDE (TRIPLE T) REPE ATS duplication CMT1A Figure 1-11 The unequal meiotic recombination (crossing- over) resulting in duplication [CMT1A] and deletion [HNPP]. 14 Several disorders are recognized to have a phenomenon of earlier age at onset of disease in successive generations. This is known as anticipation. This observation failed to secure a valid biological explanation, and had been put aside simply on the assumption of ascertainment bias for probands, or random variations in the age of onset.

However, some affected individuals experience increasingly progressive neuromuscular weakness of distal muscles of lower legs, feet, distal forearms, and hands, with onset in early teens and causing severe locomotor restrictions. An affected person usually presents late, with relative hypertrophy of the upper calf muscles, described as “inverted champagne bottle” associated with pes cavus due to wasting of the small muscles of the feet. ” Neurophysiological studies remain an essential method of differentiating the two major types of CMTD.

On a wider genomic level, mutations in the DNMT3b gene, causing the ICF (immunodeficiency, centromeric region instability, and facial anomalies) syndrome, result in deregulation of DNA methylation patterns. 5. This region contains a cluster of genes, which is susceptible to a number of epigenetic alterations, manifesting with the BWS phenotype and tumorigenesis, particularly Wilms’ tumor and other childhood embryonal tumors (Figure 1-8). , 1993). The epigenetic phenomenon is probably significant for the phenotypic manifestations in some other hereditary tumors.

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